|
1. Cobb,
H.C., Churn, S.B., Woolcott, W.S., Leftwich, F.B. and
Nolin, J.M. 1988.
Preliminary study of calcium kinetics during acute stress
in divergent life forms. Virginia Journal of Science 39(3):224-233.
2. Churn, S.B., Taft, W.C., Billingsley, M.L., Blair,
R.E. and DeLorenzo, R.J. 1990.br>
Temperature modulation of ischemia-induced neuronal cell
death and ischemia-induced inhibition of calcium/calmodulin-dependent
protein kinase II. Stroke. 21:1715-1721.
3. Churn, S.B., Anderson, W.W., and DeLorenzo, R.J. 1991.
Exposure of hippocampal slices to magnesium-free medium
produces epileptiform activity and simultaneously decreases
calcium and calmodulin-dependent protein kinase II activity.
Epilepsy Research. 9:211-217.
4. Churn, S.B., Yaghmai, A., Povlishock, J.T., Rafiq,
A. and DeLorenzo, R.J. 1992.
Global forebrain ischemia results in decreased immunoreactivity
of calcium and calmodulin-dependent protein kinase II.
Journal of Cerebral Blood Flow and Metabolism. 12:784-793.
5. Churn, S.B., Taft, W.C., Billingsley, M.S., Sankaran,
and DeLorenzo, R.J. 1992.
Global forebrain ischemia induces a post-translational
modification of multifunctional calcium and calmodulin-dependent
kinase II. Journal of Neurochemistry. 56:1221-1232.
6. Perlin, J.B., Churn, S.B., Lothman, E.W. and DeLorenzo,
R.J. 1992. Loss of type
II calcium/calmodulin-dependent kinase activity correlates
with stages of development of electrographic Seizures
in Status Epilepticus in Rat. Epilepsy Research. 11:111-118.
7. Churn, S.B., Sankaran, B., Haley, B.E. and DeLorenzo,
R.J. 1993. Ischemia results in decreased ATP affinity
of CaM kinase II. Biophysical and Biochemical Research
Communications 193:934-940.
8. Churn, S.B., Sombati, S., Taft, W.C. and DeLorenzo,
R.J. 1993. Excitotoxicity
affects membrane potential and calmodulin kinase activity
in cortical neurons. Stroke 24:271-278.
9. Churn, S.B., Sankaran, B., Haley, B.E., and DeLorenzo,
R.J. 1993. Ischemic brain injury selectively alters ATP
binding of calcium and calmodulin-dependent protein kinase
II. Biochemical and Biophysical Research Communication.
June 30; 193(3):934-40.
10. Churn, S.B., DeLorenzo, R.J. and S.M. Shapiro, S.M.
1995. Bilirubin induces a calcium-dependent inhibition
of multifunctional Ca2+/calmodulin-dependent kinase II
activity, in vitro. Pediatric Research. 38:949-954.
11. Limbrick, D.D., Churn, S.B., Sombati, S. and DeLorenzo,
R.J. 1995. Inability to restore resting intracellular
calcium levels as an early indication of delayed neuronal
cell death. Brain Research. 690:145-156.
12. Churn, S.B., Limbrick, D., Sombati, S. and DeLorenzo,
R.J. 1995. Excitotoxic activation of the NMDA glutamate
receptor results in inhibition of calcium/calmodulin kinase
II activity in cultured hippocampal neurons. Journal of
Neuroscience 15:3200-3214.
13. Parsons, J.T., Churn, S.B. and DeLorenzo. R.J. 1997.
Ischemia-induced inhibition of calcium uptake into rat
brain microsomes mediated by Mg/Ca ATPase. Journal of
Neurochemistry. 68:1124-1134.
14. Parsons, J.T., Churn, S.B. and DeLorenzo, R.J. 1998.
Separation of radiolabeled orthophosphate and adenosine
5'-triphosphate by 20% polyacrylamide gel electrophoresis:
An assay for brain microsomal Mg2+/Ca2+ ATPase activity.
Analytical Biochemistry. 264:74-81.
15. Churn, S.B. and DeLorenzo, R.J. 1998. Modulation of
GABAergic receptor binding by activation of calcium/calmodulin-dependent
kinase II membrane phosphorylation. Brain Research. 809:68-76.
16. Parsons, J.T., Churn, S.B. and DeLorenzo, R.J. 1999.
Global ischemia-induced inhibition of the coupling ratio
of calcium uptake and ATP hydrolysis by rat whole brain
microsomal Mg2+/Ca2+ ATPase. Brain Research 834:32-41.
17. Babcock, A.M., Liu, H., Paden, C.M., Churn, S.B. and
Pittman, A. 1999. In vivo
glutamate neurotoxicity is associated with a reduction
in calcium/calmodulin-
dependent protein kinase II immunoreactivity. Journal
Neuroscience Research. 56:36-43.
18. Blair, R.E., Churn, S.B., Sombati, S. and DeLorenzo,
R.J. 1999. Long-lasting decrease in neuronal Ca2+/calmodulin-dependent
protein kinase II activity associated with epileptogenesis
in a hippocampal cell culture model of epilepsy. Brain
Research. 851, 54-65.
19. Kochan, L.D., Churn, S.B., Omojokun, O., Rice, A.C.
and DeLorenzo, R.J. 1999. Status epilepticus results in
an NMDA receptor-dependent inhibition of
Ca2+/calmodulin-dependent kinase II activity in the rat.
Neuroscience. 95(3), 735-743.
20. Conlee, J.W., Shapiro, S.M. and Churn, S.B. 2000.
Delayed development of
calcium/calmodulin kinase II subunits in the cerebellum
of Gunn rats: A quantitative light microscopic analysis.
ACTA Neuropathologica, 99, 393-401.
21. Churn, S.B. Sombati, S., Jakoi, E. and DeLorenzo,
R.J. 2000. Knockdown of the alpha subunit of CaM kinase
II results in altered neuronal excitability and inhibition
of GABAA receptor function. Proceedings of the National
Academy of Science, USA. 97(10), 5604-5609.
22. Parsons, J. T., Churn, S.B., Kochan, L.D. and DeLorenzo,
R.J. 2000. Pilocarpine-induced status epilepticus causes
N-methyl-D-aspartate receptor-dependent inhibition of
microsomal Mg2+/Ca2+ ATPase-mediated Ca2+ uptake. Journal
of Neurochemistry. 75(3), 1209-1218.
23. Churn, S.B., Kochan, L.D., and DeLorenzo, R.J. 2000.
Chronic inhibition of Ca2+/calmodulin kinase II activity
in the pilocarpine model of epilepsy. Brain Research.
875, 66-77.
24. Conlee, J.W., Shapiro, S.M. and Churn, S.B. 2000.
Expression of the alpha and beta subunits of Ca2+/calmodulin
kinase II in the cerebellum of jaundiced Gunn rats during
development: a quantitative light microscopic analysis.
Acta Neuropathol (Berl). 99(4):393-401.
25. Kurz, J., Parsons, J.T., Sheets, D., Rana, A. and
Churn, S.B. 2001. A significant
increase in both basal and maximal calcineurin activity
in the rat pilocarpine model of status epilepticus. Journal
of Neurochemistry 78(2):304 315.
26. Parsons, J.T., Churn, S.B. and DeLorenzo, R.J. 2001.
Chronic inhibition of cortex
microsomal Mg2+/Ca 2++ ATPase-mediated Ca 2+ uptake in
the rat pilocarpine model following epileptogenesis. Journal
of Neurochemistry. 79, 319-327.
27. Churn, S.B., Rana, A., Lee, K., Parsons, J.T., DeBlas,
A. and DeLorenzo, R.J. 2002.
Calcium/calmodulin-dependent kinase II phosphorylation
of the GABAA receptor alpha 1 subunit modulates benzodiazepine
binding. Journal of Neurochemistry, 82, 1065-1076.
28. Kurz, J., Hamm R., Churn, S.B. Alteration of cortical
and hippocampal phosphatase
activity following head trauma in the rat. Journal of
Neurotrauma. (Manuscript revised, #3164).
29. Blair, R.E., Sombati, S., Jakoi, E.R., Gibbs, J.W.
,III, Sievert, W.L., Churn, S.B., Coulter, D.A., DeLorenzo,
R. J. Decreased GABAA receptor expression in an in vitro
hippocampal neuronal culture model of spontaneous recurrent
seizures. Journal of Neurochemistry. (Submitted, June
2002).
30. Kurz, J., Parsons, J.T., Rana, A, Churn, S.B. 2003.
Translocation and activation of calcineurin following
prolonged status epilepticus in the rat. Neurobiology
of Disease, 14:483-493.
31. Churn, S.B., Franks, P.D. and Thiessen, M. Efficacy
of Topiramate in Otherwise Refractory Status Epilepticus
in the Rat. Epilepsia (manuscript revised #00682-2003).
32. Parsons, J.T., Sun, D.A., DeLorenzo, R.J., and Churn,
S.B. Neuronal specific endoplasmic reticulum Mg2+/Ca2+ATPase
Ca2+ sequestration in mixed primary hippocampal culture
homogenates. Analytical Biochemistry (manuscript in press
# ABIO-03-68).
33. Churn, S.B., Sombati, S. DeLorenzo, R.J. Calcium and
calmodulin kinase II
regulation of agonist-evoked currents at GABAA receptors.
Neuroscience, (in preparation).
34. Singleton, M.W., Ryan, M.L., Kurz, J.B., Holbert,
W.H., and Churn, S.B. Age dependence of SE-induced inhibition
of CaM kinase II activity in the rat pilocarpine model.
Epilepsia, (In Preparation).
35. Kurz, J., Singleton R., Hamm R., Povlishock J.T. and
Churn, S.B. Traumatic brain injury-induced alteration
of synaptic calcineurin concentration. Biochemical and
Biophysical Research Communications (In preparation).
Churn, S.B., Taft, W.C.
and DeLorenzo, R.J. 1990.
Effects of ischemia on multifunctional calcium/calmodulin-dependent
protein kinase type II in the gerbil. Cerebral Vascular
Diseases: 17th Princeton Conference. Stroke 21 (suppl
111):Ill- I 12-Ill-116.
Churn, S.B. 1995. Multifunctional
calcium and calmodulin-dependent kinase II in neuronal
function and disease. Advances in Neuroimmunology 5:241-259.
Churn, S.B. and DeLorenzo,
R.J. 1996.
Intracellular messengers and mediators. Cerebrovascular
Disease: Pathophysiology, Diagnosis and Treatment. Ginsberg
MD and Bogousslavsky J. eds. Blackwell Scientific Publications,
Boston.
1. Tennes-Reese, K.A.,
Churn, S.B., Taft, W.C., Billingsley, M.L. and DeLorenzo,
R.J. 1988.
Ischemia-induced inhibition of CaM kinase
II activity in gerbil rain. Society for Neurosciences,
vol. 14 (1) p. 1 86 a. 80.9.
2. Churn, S.B., Blair,
R.E., Taft, W.C. and DeLorenzo, R.J. 1989.
Hypothennia blocks ischemia-induced inhibition
of CaM kinase II. Society for Neurosciences, vol. 15(l)
p.306 a. 149.3.
3. Perlin, J.B., Churn,
S.B., Lothman, E.W., Taft, W.C. and DeLorenzo, R.J.
1989.
Continuous hippocampal stimulation decreases
calcium calmodulin-dependent protein kinase II activity.
Society for Neurosciences, vol. 15(l) p.454 a. 181.10.
4. Anderson, W.W., Churn,
S.B. and DeLorenzo, R.J. 1989.
Decrease in calcium calmodulin-dependent protein
kinase II activity in rat hippocampal slices in magnesium-free
medium. Society for Neurosciences, Vol. 15(l) p.533
a. 336.8.
5. Churn, S.B., Taft, W.C.,
Blair, R.E. and DeLorenzo, R.J. 1990.
Calcium/calmodulin-dependent protein kinase
II isolated from control and ischemic gerbils. Society
for Neurosciences, vol. 16(l) p.278 a. II 8.9.
6. Perlin, J.B., Churn,
S.B. and DeLorenzo, R.J. 1990.
Dynamic modulation of CaM kinase II activity
in a model of status epilepticus. Molecular
Mechanisms of Cell Communication. Proceedings
of the 5th annual Aspen MD/PhD student conference.
7. Churn, S.B., Sombati,
S., Taft, W.C., Willey, A.L. and DeLorenzo, R.J. 1991.
Glutamate-induced excitotoxicity results in
decreased CaM kinase II activity in cultured rat cortical
neurons. Society for Neurosciences, vol. 17(l) p. 783
a. 314.4
8. Churn, S.B., Taft, W.C.
and DeLorenzo, R.J. 1991.
Ischemia-induced modification(s) of calcium/calmodulin
kinase II. Society for Neurochemistry, 22 p.7 a. 134.
9. Churn, S.B., Sombati,
S. and DeLorenzo, R.J. 1992.
Excitotoxic NMDA receptor activation results
in inhibition of CaM kinase II activity in rat cortical
cell culture. Society for Neurosciences, vol. 18(l)
p. 440 Abstr. 190.5.
10. Churn, S.B., Limbrick,
D. and DeLorenzo, R.J. 1993.
Possible role of excitotoxicity-induced inhibition
of CaM kinase II activity in delayed neuronal death.
Society for Neurosciences, vol 19(2) p. 1777 Abstr.
729.3.
11. Shapiro, S.M., Conlee,
J.W. and Churn, S.B. 1993.
Bilirubin toxicity selectively decreases neuronal
CaM kinase II enzyme activity. Pediatric Research, vol.
33 (4) p. 236A Abstr 1396.
12. Churn, S.B., Kochan,
L.D., Parsons, J.T. and DeLorenzo, R.J. 1994.
Status epilepticus results in altered microsomal
calcium sequestration. American Epilepsy Society vol
35(8):4.
13. Parsons, J.T., Churn,
S.B. and DeLorenzo, R.J. 1994.
Subcellular localization of ischemia-induced
inhibition of CaM kinase II. Society for Neurosciences,
vol. 20 (2) p. 1041, Abstr. 424. 1.
14. Churn, S.B., Limbrick,
D. and DeLorenzo, R.J. 1994.
Glutamate-induced inhibition of CaM kinase
IIactivity correlates with sustained intracellular calcium.
Society for Neurosciences, vol. 20 (1) page 267, Abstr.
120.8.
15. Conlee, J.W., Shapiro,
S.M. and Churn, S.B. 1994.
Development of alpha and beta CaM kinase II
in the cerebellum of jaundiced and non-jaundiced Gunn
rats. Child Neurology Society.
16. Churn, S.B., Gray,
A.M., DeLorenzo, R.J. and Shapiro, S.M. 1994.
Bilirubin exposure results in a calcium-specific
inhibition of multifunctional calcium/calmodulin- dependent
kinase II. Child Neurology Society.
17. Churn, S.B. and DeLorenzo,
R.J. 1995.
Cation dependence of excitotoxic glutamate-induced
inhibition of CaM kinase II activity. Society for Neurosciences,
vol. 21(1).
18. Rice, A.C., Churn,
S.B., Obias, V.J. and DeLorenzo, R.J. 1995.
Decreased neuronal calcium-calmodulin kinase
II immunoreactivity predicts neuronal cell death in
status epilepticus. Epilepsia, 36(4).
19. Kochan, L.D, Churn,
S.B. and DeLorenzo, R.J. 1995.
Alterations in rat brain microsomal calcium
uptake in an electrical model of status epilepticus.
Epilepsia, 36(4):64.
20. Parsons J.T., Churn,
S.B. and DeLorenzo, R.J. 1995.
Ischemia-induced inhibition of calcium uptake
into microsomes isolated for the rat whole brain. Society
forNeurosciences, vol.21(1):217.
21. Kochan, L.D., Churn,
S.B., Blair, R.E., Obias, V.J. and DeLorenzo, R.J. 1995.
Continuous hippocampal stimulation-induced
status epilepticus results in alterations in microsomal
calcium uptake. Society for Neurosciences, vol 21 (2):1968.
22. Rice, A.C., Churn,
S.B., Obias, V.J. and DeLorenzo, R.J. 1995.
Decreased calcium-calmodulin kinase II immunoreactivity
in hippocampal CA1 neurons in an electrical stimulation
model of status epilepticus. Society for Neurosciences,
vol. 21 (2).
23. Rice, A.C., Churn,
S.B., Shumate, M.D. and DeLorenzo, R.J. 1996.
Long-lasting decreased muscimol binding in
an in vivo model of limbic epilepsy. Society for Neurosciences,
22(3), p.2095. Abstr. 821.18.
24. Sombati, S., Jakoi,
E.M., Severt, L., Churn, S.B. and DeLorenzo., R.J. 1996.
Knock-down of alpha subunit of calcium/calmodulin-dependent
kinase II reduced inhibitory postsynaptic currents and
increased excitability in cultured hippocampal neurons.
Society for Neurosciences, 22(3), p.2089. Abstr. 818.17.
25. Kochan, L.D., Churn,
S.B., Rice, A.C. and DeLorenzo, R.J. 1996.
Long-term inhibition of Ca2+/calmodulin-dependent
kinase II activity in a model of partial complex (limbic)
epilepsy. Society for Neurosciences, 22(2), p.1418.
Abstr. 558.9.
26. Churn, S.B. and DeLorenzo,
R.J. 1996.
CaM kinase II-dependent phosphorylation of
synaptic plasmamembrane increases muscimol binding.
Society for Neurosciences, 22(1), p.813. Abstr. 324.3.
27. Parsons, J.T., Churn,
S.B. and DeLorenzo, R.J. 1996.
Calcium and calmodulin-dependent protein kinase
II regulation of brain microsomal calcium uptake mediated
by Mg2+/Ca2+ ATPase. Society for Neurosciences, 22(1),
p.367. Abstr. 148.17.
28. DeLorenzo, R.J., Rice,
A.C., Shumate, M.D. and Churn, S.B. 1996.
MK-801 pretreatment prevents the development
of spontaneous recurrent seizures in the pilocarpine
model of limbic epilepsy. Society for Neurosciences,
22(1), p.188. Abstr. 79.10.
29. Churn, S.B., Kochan,
L.D., Wallace, M.J., Lin, J. and Robinson, S.E. 1996.
Perinatal methadone exposure affects cyclic
AMP-dependent protein kinase and Ca2+/calmodulin-dependent
kinase II activity in the rat. FASEB J. 10:A455.
30. Churn, S.B., Kochan,
L.D. and DeLorenzo, R.J. 1997.
Status Epilepticus results in acute inhibition
of CaM kinase II activity and benzodiazepine binding
in cortical tissue. Society for Neurosciences, 23(2)
pg. 2158, Abstr 838.15.
31. Kochan, L.D., Churn,
S.B., Rice, A.C. and DeLorenzo, R.J. 1997.
Status Epilepticus produces acute and long-term
alterations in CaM kinase II activity. Society for Neurosciences,
23(2), pg 2159. Abstr 838.18.
32. Parsons, J.T., Churn,
S.B. and DeLorenzo, R.J. 1997.
Ischemic-induced inhibition of inorganic phosphate
liberated by the microsomal Mg2+/Ca2+ ATPase isolated
from rat brain. Society for Neurosciences, vol 23(1),
pg. 546. Abstr. 213.4.
33. Parsons, J.T., Churn,
S.B., & DeLorenzo, R.J. 1998.
Calcium and calmodulin-dependent protein kinase
II regulation of brain microsomal calcium uptake mediated
by Mg2+/Ca2+ ATPase. Society for Neurosciences, 22,
367.
34. Kochan, L.D., Churn,
S.B., & DeLorenzo, R.J. 1998.
Alterations in rat brain microsomal calcium
uptake in an electrical model of status epilepticus.
Epilepsia, 36, 64.
35. DeLorenzo, R.J., Rice,
A.C., Shumate, M.D., and Churn, S.B. 1998.
MK-801 pretreatment prevents the development
of spontaneous recurrent seizures in the pilocarpine
model of limbic epilepsy. Society for Neurosciences,
22, 188.
36. Churn, S.B., and DeLorenzo,
R.J.
CaM kinase II-dependent phosphorylation positively
modulates GABAA receptor binding in control synaptosomal
membrane fractions. Society for Neurosciences, Vol.
24, p. 1011, 1998.
37. Churn, S.B., Sombati,
S. and DeLorenzo, R.J. 1998.
CaM kinase II regulation of GABAA receptor
function and neuronal excitability in hippocampal neurons.
Epilepsia 39(6):17.
38. Blair, R.E., Churn,
S.B., Sombati, S. and DeLorenzo, R.J.
Decrease in CaM kinase II activity associated
with epileptogenesis. Epilepsia, 40 (7), 14, 1999.
39. Morris, T.A., Blair,
R.E., Sombati, S., Jakoi, E.R., Churn, S.B., Severt,
L., Gibbs, J.W., Coulter, D.A. and DeLorenzo, R.J. 1999.
Decreased GABAA receptor function and expression
in an in vitro hippocampal culture model of spontaneous
recurrent seizures. Epilepsia, 40 (7), 14.
40. Parsons, J.T., Churn,
S.B. and DeLorenzo, R.J. 1999.
The effect of normothermic globalischemia
on varying concentrations of substrates essential for
rat brain microsomal Mg2+/Ca2+ ATPase. Society for Neurosciences,
25:841.
41. Churn, S.B., Lee, K.,
Parsons,J.T. and DeLorenzo, R.J. 1999.
CaM kinase II regulates benzodiazepine binding
by GABAA receptor subunit phosphorylation. Society for
Neurosciences, 25:822.5.
42. Churn, S.B. and DeLorenzo,
R.J. 1999.
Modulation of GABAA receptor function by calcium/calmodulin
kinase II activation. Epilepsia, 40 (7), 40:208.
43. Rana, A., Parsons,
J.T., Lee, K., DeLorenzo, R.J. and Churn, S.B. 2000.
Ca2+/calmodulin-dependent kinase II phosphorylation
positively modulates benzodiazepine binding to GABAA
receptors in the CNS. The Seventeenth Annual Daniel
T. Watts Research Poster Symposium, Virginia Commonwealth
University.
44. Churn, S.B., Sombati,
S. and DeLorenzo, R.J. 2000.
Inhibition of CaM kinase II alpha subunit
expression results in decreased GABAA receptor function.
Society for Neurosciences, 26:621.2.
45. Parsons, J.T., Churn,
S.B. and DeLorenzo, R.J. 2000.
Persistent inhibition of Ca2+ uptake by rat
cortex microsomes in the pilocarpine model of epilepsy.
Society for Neurosciences, 26:660.18.
46. Churn, S.B., Sombati,
S. and DeLorenzo, R.J. 2000.
Reversal of status epilepticus- induced inhibition
of GABAAR function by injection of CaM kinase II. Epilepsia
41(7):5.
47. Churn, S.B., Kurz,
J.E., Rana, A., Parsons, J.T. and Sheets, D. 2001.
Synaptic translocation and activation of calcineurin
in the pilocarpine model of status epilepticus. Society for Neurosciences, 27:760.8
48. Rana,A., Parsons, J.T.,
DeLorenzo, R.J., and Churn S.B. 2001.
Modulation of calmodulin kinase II activity
in subcellular fractions isolated from forebrain homogenates
in the rat pilocarpine model of status epilepticus.
Society for Neurosciences, 27:760.9.
49. Parsons, J.T., Churn,
S.B., and DeLorenzo, R.J. 2001.
Status epilepticus inhibits Mg2+/Ca2+ ATPase-dependent
Ca uptake in microsomes from the hippocampus but not
cerebellum or thalamus. Society for Neurosciences, 27:760.10.
50. Blair, R.E., Sombati,
S., Churn, S.B. and DeLorenzo, R.J. 2001.
N-methyl-D-aspartate receptor Ca2+-dependent
decrease in CaM kinase II activity in a hippocampal
neuronal culture model of spontaneous recurrent epileptiform
dishcarges. Epilepsia 41: Epilepsia 42(7):10.
51. Kurz,J.E., Gibson,
C.J., Hamm, R.J., and Churn S.B. 2001.
Moderate fluid percussion injury increases
both basal and maximal calcineurin activity in rat forebrain
homogenates. Journal of Neurotrauma; Journal of Neurotrauma,
18(10):1127.
52. Sombati, S., Sun, D.A.,
Gibson,R.J., Wilson, M.S., Churn, S.B., DeLorenzo, R.J.
and Hamm, R.J., 2001.
Traumatic Brain injury results in decreased GABAA
receptor function. Journal of Neurotrauma: Journal
of Neurotrauma, 18(10):1181.
53. Parsons J.T., Sun, D.A., Sombati S., DeLorenzo
R.J., and Churn, S.B. 2002.
Characterization of endoplasmic reticulum Mg2+/Ca2+
ATPase mediated Ca2+ uptake in the hippocampal neuronal
culture model of status epilepticus. Epilepsia 43(suppl
7:30, 1.078).
54. Rana A., Parsons J.T., de Blas A., and Churn S.B.
2002. Ca2+/calmodulin kinase II phosphorylation of
GABAA receptor subunits is significantly inhibited
during prolonged status epilepticus. Epilepsia 43
(suppl 7:143, 2.045).
55. Churn S.B., Rana A., de Blas A., and Parsons J.T.
2002. SE-induced inhibition of calmodulin kinase II-dependent
phosphorylation of the GABAA ?1 receptor subunit.
Society for Neurosciences, 28:603.3
56. Rana, A., Franks P.D., Parsons J.T., de Blas A.,
and Churn S.B. 2002. Loss of CaM kinase II-mediated
modulation of GABAA receptor phosphorylation in status
epilepticus. GABAA receptors in cellular and network
excitability: 12th Neuropharmacology Conference, Orlando,
FL.
57. Churn, S.B., Rosen, J., Rana, A., and Shaprio,
S.M. 2002. Calcium and calmodulin-dependent protein
kinase II (CaM KII), a biological marker of bilirubin
neurotoxicity. Annals of Neurology 52(3S):S121
58. Mukherjee, D., Rana, A., Parsons, J.T. Franks,
P.D. and Churn, S.B. 2003. Pathological caspase activation
in status epilepticus. Undergraduate Biochemistry
Research Symposium. Virginia Commonwealth University.
59. Franks, P.D., Thiessen, M. Bodle-biber, M.C. and
Churn, S.B. 2003. Status epilepticus-induced inhibition
of CaM kinase II activity is blocked by topiramate
administration. Society for Neurosciences, 29;634.12.
60. Churn, S.B., Franks,
P.D. and Thiessen, M. 2003. Efficacy of topiramate
in an animal model of refractory status epilepticus.
Society for Neurosciences, 29;634.13
61. Parsons, J.T., Sun,
D.A., Delorenzo, R.J. and Churn, S.B. 2003.
N-methyl-D-aspartate receptor activation dependent
inhibition of endoplasmic reticulum Mg2+/Ca2+ ATPase
mediated Ca2+ uptake in the hippocampal culture low
Mg model of status epilepticus. Society for Neurosciences,
29;212.14.
62. Rana, A., Franks, P.D., Parsons, J.T., de Blas,
A., and Churn, S.B. 2004. CaM kinase II-mediated modulation
of GABAA receptor phosphorylation in status epilepticus.
Restorative Neurology and Neuroscience (In press).
Book Review: Advances
in Enzymology (ed. A. Meister), J. Wiley and Sons.
Reviewed for the American Society of Biologists.
ASB Bulletin, vol 43.
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